https://nova.newcastle.edu.au/vital/access/ /manager/Index ${session.getAttribute("locale")} 5 https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:22789 Wed 11 Apr 2018 14:49:49 AEST ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:20226 Wed 11 Apr 2018 11:05:32 AEST ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:41023 Wed 08 Nov 2023 09:37:41 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:36195 IO₂] = 21%), moderate‐level hypoxia (F_IO₂ = 16%) and high‐level hypoxia (F_IO₂ = 13%). Exercise comprised high‐load squats and deadlifts (5 × 5 using 80% of 1‐repetition maximum with 180‐s rest). Muscle oxygenation and activation were monitored during exercise. Metabolic stress was estimated via capillary blood sampling. Perceived fatigue and soreness were also quantified following exercise. While the hypoxic conditions appeared to affect muscle oxygenation, significant differences between conditions were only noted for maximal deoxyhaemoglobin in the deadlift (P = 0·009). Blood lactate concentration increased from 1·1 to 1·2 mmol l⁻¹ at baseline to 9·5–9·8 mmol l⁻¹ after squats and 10·4–10·5 mmol l⁻¹ after deadlifts (P≤0·001), although there were no between‐condition differences. Perceived fatigue and muscle soreness were significantly elevated immediately and at 24 h following exercise, respectively, by similar magnitudes in all conditions (P≤0·001). Muscle activation did not differ between conditions. While metabolic stress is thought to moderate muscle activation and subsequent muscular development during hypoxic resistance training, it is not augmented during traditional high‐load exercise. This may be explained by the low number of repetitions performed and the long interset rest periods employed during this training. These findings suggest that high‐load resistance training might not benefit from additional hypoxia as has been shown for low‐ and moderate‐load training.]]> Thu 27 Feb 2020 13:33:36 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:27112 Sat 24 Mar 2018 07:41:36 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:32764 IO₂] = 21%) and moderate-level hypoxia (MH; F_IO₂ = 16%). Exercise comprised 3 sets of 10 repetitions of squats and deadlifts at 60% of 1 repetition maximum, with 60-second interset rest. Blood lactate (BLa⁻) was quantified after each exercise, whereas arterial oxygen saturation and heart rate (HR) were assessed after each set. Thigh circumference was measured before and after exercise. Muscle activation and oxygenation were monitored by surface electromyography (EMG) and near-infrared spectroscopy, respectively. Relative BLa⁻ concentrations were significantly higher following squats (p = 0.041) and deadlifts (p = 0.002) in MH than NORM. Arterial oxygen saturation was lower after each set in MH compared with NORM (p , 0.001), although HR and thigh circumference were not different between conditions. Integrated EMG was higher in MH than in NORM for the squat during several repetitions (p ≤ 0.032). Measures of muscle oxygen status were not significantly different between conditions (p ≥ 0.247). The main findings from this study suggest that hypoxia during moderate-load resistance exercise augments metabolite accumulation and muscle activation. However, a significant hypoxic dose was not measured at the muscle, possibly because of the moderate level of hypoxia used. The current data support previous hypotheses that have suggested hypoxia can augment some physiological responses that are important for muscular development, and may therefore provide benefit over the equivalent training in normoxia.]]> Mon 23 Jul 2018 12:49:19 AEST ]]>